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Most organisms, including humans, have parasitic DNA fragments called ‘jumping genes’ that insert themselves into DNA molecules, disrupting genetic instructions in the process. And that phenomenon can result in age-related diseases such as cancer. But researchers now report that the ‘jumping genes’ in mice become active as the mice age when a multi-function protein stops keeping them in check in order to take on another role.

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    @arilen Maybe if there was less damage (ie. we get are micronutrients) for Sirt6 to repair, it could prevent the jumping genes.

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      great summary title! thanks for submitting, @arilen