@rhonda, I would have concerns that the residual TiO2 might be worse than the BPA. Report:
https://news.utexas.edu/2018/06/20/link-found-between-type-2-diabetes-and-common-white-pigment
on a paper:
https://pubs.acs.org/doi/full/10.1021/acs.chemrestox.8b00047
Previous studies showed TiO2 inhale or ingested ended up in the blood stream where it was phagocytosed. From the paper:
“By binding to and activating the NLRP3(NALP3) inflammasome,(12,31−33) a macromolecular complex found in macrophages, neutrophils, platelets, and microglia, the TiO2 crystals cause inflammatory cascades leading to death of proximal cells.”
Furthermore, the paper itself reported an association of TiO2 crystals in the pancreases of type 2 diabetics, but not in non-diabetics. Small study – but all 8 T2D pancreases examined had the crystals whereas none of the 5 non-T2D pancreases did.
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@rhonda, I would have concerns that the residual TiO2 might be worse than the BPA. Report: https://news.utexas.edu/2018/06/20/link-found-between-type-2-diabetes-and-common-white-pigment on a paper: https://pubs.acs.org/doi/full/10.1021/acs.chemrestox.8b00047 Previous studies showed TiO2 inhale or ingested ended up in the blood stream where it was phagocytosed. From the paper: “By binding to and activating the NLRP3(NALP3) inflammasome,(12,31−33) a macromolecular complex found in macrophages, neutrophils, platelets, and microglia, the TiO2 crystals cause inflammatory cascades leading to death of proximal cells.” Furthermore, the paper itself reported an association of TiO2 crystals in the pancreases of type 2 diabetics, but not in non-diabetics. Small study – but all 8 T2D pancreases examined had the crystals whereas none of the 5 non-T2D pancreases did.